The renal tubules show varying degrees of vacuolar degeneration, necrosis and cast formation. 肾小管呈现空泡变、坏死及管型形成。
Results The adhesion, disturbance, lodge, and squamous metaplasia of cilia in tracheal epithelium; and vacuolar degeneration in epithelium cells were observed. 结果气管上皮纤毛粘连、乱、伏及鳞状化生,上皮细胞空泡变性,并可见新生的纤毛上皮细胞。
Pathologic examination revealed that hippocampal neurons displayed local ischemic damage, cells in granular layer showed severe edema, degeneration, as well as necrosis, and neuronal mitochondria showed vacuolar degeneration. 病理形态学显示神经元呈局部缺血性改变,颗粒层细胞高度水肿、变性、坏死,神经元线粒体空泡化现象。
Under an electron microscope it was observed that the cytoplasm of KC in the GC+ DENA group contained a large number of electron dense granules or KC showed vacuolar degeneration or necrosis; 电镜下证实:GC+DENA组肝窦内枯否细胞(KC)胞浆内含有大量电子致密物或细胞空泡变性及坏死;
Histopathology transformations: Liver, vacuolar degeneration and swollen in hepatocytes, small bile ducts proliferated. 组织病理学变化:肝脏,肝细胞空泡变性,胆管增生。肝小叶内和汇管区有小灶性的浆细胞浸润和嗜酸性粒细胞浸润,及后期肝细胞极度肿胀;
The skin showed eosinophilic necrosis of epidermal cells, vacuolar degeneration of epidermal basal cells, epidermal cleft, bulla and lymphocytic infiltration of the dermis. 皮肤表现为表皮细胞嗜酸性坏死和基底层细胞空泡性变,表皮发生裂隙、大泡形成以及真皮淋巴细胞浸润。
The hepatic cells and the epithelial cells of renal tubules were vacuolar degeneration, tracheal mucosa epithelial layer arterial hyperemia and hemorrhage. 组织学病变表现为脑神经空泡化,肺泡内出血和水肿,肝细胞与肾小管上皮细胞明显水泡变性,气管粘膜上皮层下高度充血、出血。
Results In the infected model group, there were obvious swelling, karyopyknosis and destruction of the structure in the brain cells, as well as myelin sheath solution and vacuolar degeneration in the mitochondrion and crest were destroyed. 结果电镜下感染模型组小鼠脑细胞胞浆水肿明显.核仁固缩,核内结构破坏,多数线粒体呈空泡样改变,核仁内可见病毒颗粒内脊破坏、髓鞘严重松解、破坏。
After 24 hours, hepatic cells swelled and occurred vacuolar degeneration and punctiform necrosis. 保存24h后,肝细胞出现肿胀、空泡变性及点状坏死,肝窦内皮细胞肿胀、变性,并开始向窦腔内脱落。
Results There were swelling, vacuolar degeneration and increased density of mitochondria, dissociated ribosomes in the cytoplasm, and dilation of rough endoplasmic reticulum and Golgi's apparatus. 结果艾滋病患者胃黏膜细胞的线粒体肿胀、空泡变性和密度增高,细胞质里有解聚的核糖体,粗面内质网、高尔基体有不同程度的扩张。
Vacuolar degeneration in the renal tubular epithelium and the liver cells; 肾小管上皮细胞空泡变性,肾小球萎缩;
The liver showed edema, vacuolar degeneration and foci necrosis. 肝水肿,狄氏间隙增宽,肝细胞空泡变性及坏死;
Compared with the spinal cord injury group, there was the mild swelling of spinal cord tissues, slightly bleeding, reduction of vacuolization of endochylema and karyopycnosis, hydrosarca of white matter axiscylinder, abatement of vacuolar degeneration in the methylprednisolone in prophylaxis group. 与脊髓损伤模型组比较,甲基强的松龙组脊髓组织肿胀较轻,出血较少,胞浆空泡化及核固缩现象减少;白质轴突水肿、空泡变性减轻。
At histopathological level, the hepatocytes, epithelial cells of renal tubule and cardiac muscle cells mainly showed granular and vacuolar degeneration. 肝细胞、肾小管上皮细胞、心肌细胞颗粒变性和空泡变性;
The lesions of pancreas were mitochondria swelling, vacuolar degeneration, endoplasmic reticulum exceedingly distending, and some fatty lipid-like material could be found in the cytoplasm. 胰脏主要表现在胰腺细胞胞浆内线粒体肿胀,空泡样变,内质网极度扩张,胞浆内脂质样物质聚集。
The results showed that: ① major pathological changes was fatty and vacuolar degeneration of hepatocytes, cloudy swelling of renal epithelial cells and myocardial cells; 结果表明:①青霉酸中毒尼西鸡的主要病理变化是肝细胞脂肪变性、肾小管上皮细胞浊肿、心肌细胞颗粒变性;
The mitochondrium in the ischemia/ reperfusion model group appeared vacuolar degeneration. 脑缺血再灌注模型组线粒体呈空泡样变;
Histopathology showed hyperkeratosis accompanied by focal parakeratosis, vacuolar degeneration of basal layer and superficial dermal bandlike infiltration composed of lymphocytes, and also eosinophil and plasma cells. 组织病理改变主要为角化过度伴灶状角化不全,基底层液化变性,真皮浅层可见淋巴细胞大致呈带状分布,并可见嗜酸性粒细胞和浆细胞浸润。
Epidermal changes consist of acanthosis, vacuolar degeneration, parakeratosis, hyperkeratosis and necrosis. 上皮的变化包括棘细胞增生,空泡变性,不全角化,过度角化和坏死等。
The mitochondrion of vacuolar degeneration represents ubiquity in colorectal adenocarcinoma, colorectal adenoma and normal colorectal mucosa and its configuration even tends to rotundity. 线粒体空泡化普遍存在于大肠腺癌、腺瘤和正常组织中,其形态更趋向于圆形。
Pathologically, hepatocytes in high Mo groups showed granular degeneration and vacuolar degeneration. 病理形态学观察,高钼组雏鸡肝细胞呈现不同程度的颗粒变性和空泡变性。
In group DM glomerular basement membrane thickening, a large number of mesangium matrix deposition, vacuolar degeneration of tubular epithelial cells, renal interstitial fibrosis and mononuclear cells and lymphoid cell infiltration appears. 糖尿病组大鼠肾小球毛细血管基底膜增厚,系膜区基质大量沉积,肾小管上皮细胞空泡样变性,肾间质出现纤维化及单核细胞和淋巴样细胞浸润。
There were vacuolar degeneration in renal tubular cells and the tubular lumens were narrowed. 肾小管出现细胞空泡样变性,管腔变窄。
Obvious liver structure of rats in model group, hepatic lobule structure was not clear, liver cells arranged in disorder, liver cell degeneration, necrosis, hepatic periportal inflammatory cell infiltration, liver cell diffuse vacuolar degeneration. 模型组大鼠肝脏结构破坏较明显,肝小叶结构欠清,肝细胞索排列紊乱,肝细胞变性、溶解坏死、肝汇管区炎性细胞浸润明显,肝细胞呈弥漫性空泡样变。
Capillary basement membrane thickened locally, foot processes fusion of visceral epithelial cells were found in varying degrees, accompanied by vacuolar degeneration of some mitochondrial. 毛细血管基底膜局部增厚,肾小球脏层上皮细胞出现不同程度的足突融合现象并伴有部分线粒体空泡变性。
The renal tubular epithelial cells were vacuolar degeneration, necrosis. There were cell debris and tube-type in the lumen of renal tubular. M组肾小球变形皱缩,肾小管上皮细胞空泡变性、细胞片状坏死,肾小管管腔变窄,其内出现细胞碎片或管型形成,肾间质可见出血和炎性细胞浸润。
Pathologic changes: In group A, the tissue structure is clear without edema, vacuolar degeneration, cloudy swelling and necrosis. 病理观察:A组各组织结构清晰,未见水肿、空泡变性、浊肿及坏死。
Vacuolar degeneration was checked in brain, heart, kidney, spleen and ballocks or ovaries. 病理组织学观察可见脑、心脏、肾、脾、睾丸和卵巢等组织发生明显空泡变性。
Jiawei Yinchenhao Decoction low dose group of rat liver tissue structure failure, liver cell degeneration, necrosis, inflammatory cell run lighter than in the model group, showed moderate diffuse vacuolar degeneration. 加味茵陈蒿汤低剂量组大鼠肝组织结构部分破坏,肝细胞变性、溶解坏死、炎性细胞润较模型组轻,呈中度弥漫性空泡样变。